Ecg interpretations. How to analyze a rhythm. Normal sinus rhythm. Heart arrhythmias. Diagnosing a myocardial infarction
Содержание
- 2. Course Objectives To recognize the normal rhythm of the heart - “Normal Sinus Rhythm.” To recognize
- 3. Learning Modules ECG Basics How to Analyze a Rhythm Normal Sinus Rhythm Heart Arrhythmias Diagnosing a
- 4. Normal Impulse Conduction Sinoatrial node AV node Bundle of His Bundle Branches Purkinje fibers
- 5. Impulse Conduction & the ECG Sinoatrial node AV node Bundle of His Bundle Branches Purkinje fibers
- 6. The “PQRST” P wave - Atrial depolarization T wave - Ventricular repolarization QRS - Ventricular depolarization
- 7. The PR Interval Atrial depolarization + delay in AV junction (AV node/Bundle of His) (delay allows
- 8. Pacemakers of the Heart SA Node - Dominant pacemaker with an intrinsic rate of 60 -
- 9. The ECG Paper Horizontally One small box - 0.04 s One large box - 0.20 s
- 10. The ECG Paper (cont) Every 3 seconds (15 large boxes) is marked by a vertical line.
- 11. ECG Rhythm Interpretation Really Very Easy How to Analyze a Rhythm
- 12. Rhythm Analysis Step 1: Calculate rate. Step 2: Determine regularity. Step 3: Assess the P waves.
- 13. Step 1: Calculate Rate Option 1 Count the # of R waves in a 6 second
- 14. Step 1: Calculate Rate Option 2 Find a R wave that lands on a bold line.
- 15. Step 1: Calculate Rate Option 2 (cont) Memorize the sequence: 300 - 150 - 100 -
- 16. Step 2: Determine regularity Look at the R-R distances (using a caliper or markings on a
- 17. Step 3: Assess the P waves Are there P waves? Do the P waves all look
- 18. Step 4: Determine PR interval Normal: 0.12 - 0.20 seconds. (3 - 5 boxes) Interpretation? 0.12
- 19. Step 5: QRS duration Normal: 0.04 - 0.12 seconds. (1 - 3 boxes) Interpretation? 0.08 seconds
- 20. Rhythm Summary Rate 90-95 bpm Regularity regular P waves normal PR interval 0.12 s QRS duration
- 21. NSR Parameters Rate 60 - 100 bpm Regularity regular P waves normal PR interval 0.12 -
- 22. Arrhythmia Formation Arrhythmias can arise from problems in the: Sinus node Atrial cells AV junction Ventricular
- 23. SA Node Problems The SA Node can: fire too slow fire too fast Sinus Bradycardia Sinus
- 24. Atrial Cell Problems Atrial cells can: fire occasionally from a focus fire continuously due to a
- 25. Atrial Cell Problems Atrial cells can also: • fire continuously from multiple foci or fire continuously
- 26. Teaching Moment Multiple micro re-entrant “wavelets” refers to wandering small areas of activation which generate fine
- 27. AV Junctional Problems The AV junction can: fire continuously due to a looping re-entrant circuit block
- 28. Ventricular Cell Problems Ventricular cells can: fire occasionally from 1 or more foci fire continuously from
- 29. Arrhythmias Sinus Rhythms Premature Beats Supraventricular Arrhythmias Ventricular Arrhythmias AV Junctional Blocks
- 30. Sinus Rhythms Sinus Bradycardia Sinus Tachycardia Sinus Arrest Normal Sinus Rhythm
- 31. Rhythm #1 30 bpm Rate? Regularity? regular normal 0.10 s P waves? PR interval? 0.12 s
- 32. Sinus Bradycardia Deviation from NSR - Rate
- 33. Sinus Bradycardia Etiology: SA node is depolarizing slower than normal, impulse is conducted normally (i.e. normal
- 34. Rhythm #2 130 bpm Rate? Regularity? regular normal 0.08 s P waves? PR interval? 0.16 s
- 35. Sinus Tachycardia Deviation from NSR - Rate > 100 bpm
- 36. Sinus Tachycardia Etiology: SA node is depolarizing faster than normal, impulse is conducted normally. Remember: sinus
- 37. Sinus Arrest Etiology: SA node fails to depolarize and no compensatory mechanisms take over Sinus arrest
- 38. Premature Beats Premature Atrial Contractions (PACs) Premature Ventricular Contractions (PVCs)
- 39. Rhythm #3 70 bpm Rate? Regularity? occasionally irreg. 2/7 different contour 0.08 s P waves? PR
- 40. Premature Atrial Contractions Deviation from NSR These ectopic beats originate in the atria (but not in
- 41. Premature Atrial Contractions Etiology: Excitation of an atrial cell forms an impulse that is then conducted
- 42. Teaching Moment When an impulse originates anywhere in the atria (SA node, atrial cells, AV node,
- 43. Rhythm #4 60 bpm Rate? Regularity? occasionally irreg. none for 7th QRS 0.08 s (7th wide)
- 44. PVCs Deviation from NSR Ectopic beats originate in the ventricles resulting in wide and bizarre QRS
- 45. PVCs Etiology: One or more ventricular cells are depolarizing and the impulses are abnormally conducting through
- 46. Teaching Moment When an impulse originates in a ventricle, conduction through the ventricles will be inefficient
- 47. Ventricular Conduction Normal Signal moves rapidly through the ventricles Abnormal Signal moves slowly through the ventricles
- 48. Supraventricular Arrhythmias Atrial Fibrillation Atrial Flutter Paroxysmal Supra Ventricular Tachycardia (PSVT)
- 49. Rhythm #5 100 bpm Rate? Regularity? irregularly irregular none 0.06 s P waves? PR interval? none
- 50. Atrial Fibrillation Deviation from NSR No organized atrial depolarization, so no normal P waves (impulses are
- 51. Atrial Fibrillation Etiology: due to multiple re-entrant wavelets conducted between the R & L atria and
- 52. Rhythm #6 70 bpm Rate? Regularity? regular flutter waves 0.06 s P waves? PR interval? none
- 53. Atrial Flutter Deviation from NSR No P waves. Instead flutter waves (note “sawtooth” pattern) are formed
- 54. Atrial Flutter Etiology: Reentrant pathway in the right atrium with every 2nd, 3rd or 4th impulse
- 55. Rhythm #7 74 ?148 bpm Rate? Regularity? Regular ? regular Normal ? none 0.08 s P
- 56. PSVT: Paroxysmal Supra Ventricular Tachycardia Deviation from NSR The heart rate suddenly speeds up, often triggered
- 57. AV Nodal Blocks 1st Degree AV Block 2nd Degree AV Block, Type I 2nd Degree AV
- 58. Rhythm #10 60 bpm Rate? Regularity? regular normal 0.08 s P waves? PR interval? 0.36 s
- 59. 1st Degree AV Block Deviation from NSR PR Interval > 0.20 s
- 60. 1st Degree AV Block Etiology: Prolonged conduction delay in the AV node or Bundle of His.
- 61. Rhythm #11 50 bpm Rate? Regularity? regularly irregular nl, but 4th no QRS 0.08 s P
- 62. 2nd Degree AV Block, Type I Deviation from NSR PR interval progressively lengthens, then the impulse
- 63. 2nd Degree AV Block, Type I Etiology: Each successive atrial impulse encounters a longer and longer
- 64. Rhythm #12 40 bpm Rate? Regularity? regular nl, 2 of 3 no QRS 0.08 s P
- 65. 2nd Degree AV Block, Type II Deviation from NSR Occasional P waves are completely blocked (P
- 66. Rhythm #13 40 bpm Rate? Regularity? regular no relation to QRS wide (> 0.12 s) P
- 67. 3rd Degree AV Block Deviation from NSR The P waves are completely blocked in the AV
- 68. 3rd Degree AV Block Etiology: There is complete block of conduction in the AV junction, so
- 69. Remember When an impulse originates in a ventricle, conduction through the ventricles will be inefficient and
- 70. Ventricular Fibrillation Rhythm: irregular-coarse or fine, wave form varies in size and shape Fires continuously from
- 71. Ventricular Tachycardia Ventricular cells fire continuously due to a looping re-entrant circuit Rate usually regular, 100
- 72. Asystole Ventricular standstill, no electrical activity, no cardiac output – no pulse! Cardiac arrest, may follow
- 73. IdioVentricular Rhythm Escape rhythm (safety mechanism) to prevent ventricular standstill HIS/purkinje system takes over as the
- 74. Diagnosing a MI To diagnose a myocardial infarction you need to go beyond looking at a
- 75. The 12-Lead ECG The 12-Lead ECG sees the heart from 12 different views. Therefore, the 12-Lead
- 76. The 12-Leads The 12-leads include: 3 Limb leads (I, II, III) 3 Augmented leads (aVR, aVL,
- 77. Views of the Heart Some leads get a good view of the: Anterior portion of the
- 78. ST Elevation One way to diagnose an acute MI is to look for elevation of the
- 79. ST Elevation (cont) Elevation of the ST segment (greater than 1 small box) in 2 leads
- 80. Anterior View of the Heart The anterior portion of the heart is best viewed using leads
- 81. Anterior Myocardial Infarction If you see changes in leads V1 - V4 that are consistent with
- 82. Putting it all Together Do you think this person is having a myocardial infarction. If so,
- 83. Interpretation Yes, this person is having an acute anterior wall myocardial infarction.
- 84. Other MI Locations Now that you know where to look for an anterior wall myocardial infarction
- 85. Views of the Heart Some leads get a good view of the: Anterior portion of the
- 86. Other MI Locations Second, remember that the 12-leads of the ECG look at different portions of
- 87. Other MI Locations Now, using these 3 diagrams let’s figure where to look for a lateral
- 88. Anterior MI Remember the anterior portion of the heart is best viewed using leads V1- V4.
- 89. Lateral MI So what leads do you think the lateral portion of the heart is best
- 90. Inferior MI Now how about the inferior portion of the heart? Limb Leads Augmented Leads Precordial
- 91. Putting it all Together Now, where do you think this person is having a myocardial infarction?
- 92. Inferior Wall MI This is an inferior MI. Note the ST elevation in leads II, III
- 93. Putting it all Together How about now?
- 94. Anterolateral MI This person’s MI involves both the anterior wall (V2-V4) and the lateral wall (V5-V6,
- 95. Reading 12-Lead ECGs The best way to read 12-lead ECGs is to develop a step-by-step approach
- 96. Rate Rhythm Axis Intervals Hypertrophy Infarct In Module II you learned how to calculate the rate.
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