Ecg interpretations. How to analyze a rhythm. Normal sinus rhythm. Heart arrhythmias. Diagnosing a myocardial infarction

Содержание

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Course Objectives To recognize the normal rhythm of the heart -

Course Objectives

To recognize the normal rhythm of the heart - “Normal

Sinus Rhythm.”
To recognize the 17 most common rhythm disturbances (3-Lead)
To be shown an acute myocardial infarction on a 12-Lead ECG.
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Learning Modules ECG Basics How to Analyze a Rhythm Normal Sinus

Learning Modules

ECG Basics
How to Analyze a Rhythm
Normal Sinus Rhythm
Heart Arrhythmias
Diagnosing a

Myocardial Infarction
Advanced 12-Lead Interpretation
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Normal Impulse Conduction Sinoatrial node AV node Bundle of His Bundle Branches Purkinje fibers

Normal Impulse Conduction

Sinoatrial node
AV node
Bundle of His
Bundle Branches
Purkinje fibers

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Impulse Conduction & the ECG Sinoatrial node AV node Bundle of His Bundle Branches Purkinje fibers

Impulse Conduction & the ECG

Sinoatrial node
AV node
Bundle of His
Bundle Branches
Purkinje fibers

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The “PQRST” P wave - Atrial depolarization T wave - Ventricular repolarization QRS - Ventricular depolarization

The “PQRST”

P wave - Atrial depolarization

T wave - Ventricular repolarization

QRS - Ventricular depolarization
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The PR Interval Atrial depolarization + delay in AV junction (AV

The PR Interval

Atrial depolarization
+
delay in AV junction
(AV node/Bundle of

His)
(delay allows time for the atria to contract before the ventricles contract)
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Pacemakers of the Heart SA Node - Dominant pacemaker with an

Pacemakers of the Heart

SA Node - Dominant pacemaker with an intrinsic

rate of 60 - 100 beats/ minute.
AV Node - Back-up pacemaker with an intrinsic rate of 40 - 60 beats/minute.
Ventricular cells - Back-up pacemaker with an intrinsic rate of 20 - 45 bpm.
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The ECG Paper Horizontally One small box - 0.04 s One

The ECG Paper

Horizontally
One small box - 0.04 s
One large box -

0.20 s
Vertically
One large box - 0.5 mV
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The ECG Paper (cont) Every 3 seconds (15 large boxes) is

The ECG Paper (cont)
Every 3 seconds (15 large boxes) is marked

by a vertical line.
This helps when calculating the heart rate.
NOTE: the following strips are not marked but all are 6 seconds long.

3 sec

3 sec

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ECG Rhythm Interpretation Really Very Easy How to Analyze a Rhythm

ECG Rhythm Interpretation

Really Very Easy How to Analyze a Rhythm

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Rhythm Analysis Step 1: Calculate rate. Step 2: Determine regularity. Step

Rhythm Analysis
Step 1: Calculate rate.
Step 2: Determine regularity.
Step 3: Assess the P waves.
Step 4: Determine

PR interval.
Step 5: Determine QRS duration.
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Step 1: Calculate Rate Option 1 Count the # of R

Step 1: Calculate Rate
Option 1
Count the # of R waves in

a 6 second rhythm strip, then multiply by 10.
Reminder: all rhythm strips in the Modules are 6 seconds in length.
Interpretation?

9 x 10 = 90 bpm

3 sec

3 sec

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Step 1: Calculate Rate Option 2 Find a R wave that

Step 1: Calculate Rate
Option 2
Find a R wave that lands

on a bold line.
Count the number of large boxes to the next R wave. If the second R wave is 1 large box away the rate is 300, 2 boxes - 150, 3 boxes - 100, 4 boxes - 75, etc. (cont)

R wave

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Step 1: Calculate Rate Option 2 (cont) Memorize the sequence: 300

Step 1: Calculate Rate
Option 2 (cont)
Memorize the sequence:
300 - 150

- 100 - 75 - 60 - 50
Interpretation?

300

150

100

75

60

50

Approx. 1 box less than 100 = 95 bpm

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Step 2: Determine regularity Look at the R-R distances (using a

Step 2: Determine regularity
Look at the R-R distances (using a caliper

or markings on a pen or paper).
Regular (are they equidistant apart)? Occasionally irregular? Regularly irregular? Irregularly irregular?
Interpretation?

Regular

R

R

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Step 3: Assess the P waves Are there P waves? Do

Step 3: Assess the P waves
Are there P waves?
Do the P

waves all look alike?
Do the P waves occur at a regular rate?
Is there one P wave before each QRS?
Interpretation?

Normal P waves with 1 P wave for every QRS

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Step 4: Determine PR interval Normal: 0.12 - 0.20 seconds. (3

Step 4: Determine PR interval
Normal: 0.12 - 0.20 seconds.
(3 -

5 boxes)
Interpretation?

0.12 seconds

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Step 5: QRS duration Normal: 0.04 - 0.12 seconds. (1 - 3 boxes) Interpretation? 0.08 seconds

Step 5: QRS duration
Normal: 0.04 - 0.12 seconds.
(1 - 3

boxes)
Interpretation?

0.08 seconds

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Rhythm Summary Rate 90-95 bpm Regularity regular P waves normal PR

Rhythm Summary
Rate 90-95 bpm
Regularity regular
P waves normal
PR interval 0.12 s
QRS duration 0.08 s
Interpretation?

Normal Sinus Rhythm

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NSR Parameters Rate 60 - 100 bpm Regularity regular P waves

NSR Parameters

Rate 60 - 100 bpm
Regularity regular
P waves normal
PR interval 0.12 - 0.20 s
QRS duration 0.04

- 0.12 s
Any deviation from above is sinus tachycardia, sinus bradycardia or an arrhythmia
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Arrhythmia Formation Arrhythmias can arise from problems in the: Sinus node

Arrhythmia Formation

Arrhythmias can arise from problems in the:
Sinus node
Atrial cells
AV junction
Ventricular

cells
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SA Node Problems The SA Node can: fire too slow fire

SA Node Problems

The SA Node can:
fire too slow
fire too fast
Sinus Bradycardia
Sinus

Tachycardia*

*Sinus Tachycardia may be an appropriate response to stress.

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Atrial Cell Problems Atrial cells can: fire occasionally from a focus

Atrial Cell Problems

Atrial cells can:
fire occasionally from a focus
fire continuously

due to a looping re-entrant circuit
Premature Atrial Contractions (PACs)
Atrial Flutter
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Atrial Cell Problems Atrial cells can also: • fire continuously from

Atrial Cell Problems

Atrial cells can also:
• fire continuously from multiple foci


or
fire continuously due to multiple micro re-entrant “wavelets”
Atrial Fibrillation
Atrial Fibrillation
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Teaching Moment Multiple micro re-entrant “wavelets” refers to wandering small areas

Teaching Moment

Multiple micro re-entrant “wavelets” refers to wandering small areas of

activation which generate fine chaotic impulses. Colliding wavelets can, in turn, generate new foci of activation.

Atrial tissue

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AV Junctional Problems The AV junction can: fire continuously due to

AV Junctional Problems

The AV junction can:
fire continuously due to a looping

re-entrant circuit
block impulses coming from the SA Node
Paroxysmal Supraventricular Tachycardia
AV Junctional Blocks
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Ventricular Cell Problems Ventricular cells can: fire occasionally from 1 or

Ventricular Cell Problems

Ventricular cells can:
fire occasionally from 1 or more foci
fire

continuously from multiple foci
fire continuously due to a looping re-entrant circuit
Premature Ventricular Contractions (PVCs)
Ventricular Fibrillation
Ventricular Tachycardia
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Arrhythmias Sinus Rhythms Premature Beats Supraventricular Arrhythmias Ventricular Arrhythmias AV Junctional Blocks

Arrhythmias

Sinus Rhythms
Premature Beats
Supraventricular Arrhythmias
Ventricular Arrhythmias
AV Junctional Blocks

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Sinus Rhythms Sinus Bradycardia Sinus Tachycardia Sinus Arrest Normal Sinus Rhythm

Sinus Rhythms

Sinus Bradycardia
Sinus Tachycardia
Sinus Arrest
Normal Sinus Rhythm

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Rhythm #1 30 bpm Rate? Regularity? regular normal 0.10 s P

Rhythm #1

30 bpm

Rate?

Regularity?

regular

normal

0.10 s

P waves?

PR interval?

0.12 s

QRS duration?

Interpretation?

Sinus Bradycardia

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Sinus Bradycardia Deviation from NSR - Rate

Sinus Bradycardia
Deviation from NSR
- Rate < 60 bpm

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Sinus Bradycardia Etiology: SA node is depolarizing slower than normal, impulse

Sinus Bradycardia
Etiology: SA node is depolarizing slower than normal, impulse is

conducted normally (i.e. normal PR and QRS interval).
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Rhythm #2 130 bpm Rate? Regularity? regular normal 0.08 s P

Rhythm #2

130 bpm

Rate?

Regularity?

regular

normal

0.08 s

P waves?

PR interval?

0.16 s

QRS duration?

Interpretation?

Sinus Tachycardia

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Sinus Tachycardia Deviation from NSR - Rate > 100 bpm

Sinus Tachycardia
Deviation from NSR
- Rate > 100 bpm

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Sinus Tachycardia Etiology: SA node is depolarizing faster than normal, impulse

Sinus Tachycardia
Etiology: SA node is depolarizing faster than normal, impulse is

conducted normally.
Remember: sinus tachycardia is a response to physical or psychological stress, not a primary arrhythmia.
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Sinus Arrest Etiology: SA node fails to depolarize and no compensatory

Sinus Arrest
Etiology: SA node fails to depolarize and no compensatory mechanisms

take over
Sinus arrest is usually a transient pause in sinus node activity
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Premature Beats Premature Atrial Contractions (PACs) Premature Ventricular Contractions (PVCs)

Premature Beats

Premature Atrial Contractions (PACs)
Premature Ventricular Contractions (PVCs)

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Rhythm #3 70 bpm Rate? Regularity? occasionally irreg. 2/7 different contour

Rhythm #3

70 bpm

Rate?

Regularity?

occasionally irreg.

2/7 different contour

0.08 s

P waves?

PR interval?

0.14 s (except 2/7)

QRS duration?

Interpretation?

NSR with Premature Atrial Contractions

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Premature Atrial Contractions Deviation from NSR These ectopic beats originate in

Premature Atrial Contractions
Deviation from NSR
These ectopic beats originate in the atria

(but not in the SA node), therefore the contour of the P wave, the PR interval, and the timing are different than a normally generated pulse from the SA node.
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Premature Atrial Contractions Etiology: Excitation of an atrial cell forms an

Premature Atrial Contractions
Etiology: Excitation of an atrial cell forms an impulse

that is then conducted normally through the AV node and ventricles.
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Teaching Moment When an impulse originates anywhere in the atria (SA

Teaching Moment

When an impulse originates anywhere in the atria (SA node,

atrial cells, AV node, Bundle of His) and then is conducted normally through the ventricles, the QRS will be narrow (0.04 - 0.12 s).
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Rhythm #4 60 bpm Rate? Regularity? occasionally irreg. none for 7th

Rhythm #4

60 bpm

Rate?

Regularity?

occasionally irreg.

none for 7th QRS

0.08 s (7th

wide)

P waves?

PR interval?

0.14 s

QRS duration?

Interpretation?

Sinus Rhythm with 1 PVC

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PVCs Deviation from NSR Ectopic beats originate in the ventricles resulting

PVCs
Deviation from NSR
Ectopic beats originate in the ventricles resulting in wide

and bizarre QRS complexes.
When there are more than 1 premature beats and look alike, they are called “uniform”. When they look different, they are called “multiform”.
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PVCs Etiology: One or more ventricular cells are depolarizing and the

PVCs
Etiology: One or more ventricular cells are depolarizing and the impulses

are abnormally conducting through the ventricles.
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Teaching Moment When an impulse originates in a ventricle, conduction through

Teaching Moment

When an impulse originates in a ventricle, conduction through the

ventricles will be inefficient and the QRS will be wide and bizarre.
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Ventricular Conduction Normal Signal moves rapidly through the ventricles Abnormal Signal moves slowly through the ventricles

Ventricular Conduction

Normal
Signal moves rapidly through the ventricles

Abnormal
Signal moves slowly through the

ventricles
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Supraventricular Arrhythmias Atrial Fibrillation Atrial Flutter Paroxysmal Supra Ventricular Tachycardia (PSVT)

Supraventricular Arrhythmias

Atrial Fibrillation
Atrial Flutter
Paroxysmal Supra Ventricular Tachycardia (PSVT)

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Rhythm #5 100 bpm Rate? Regularity? irregularly irregular none 0.06 s

Rhythm #5

100 bpm

Rate?

Regularity?

irregularly irregular

none

0.06 s

P waves?

PR interval?

none

QRS duration?

Interpretation?

Atrial Fibrillation

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Atrial Fibrillation Deviation from NSR No organized atrial depolarization, so no

Atrial Fibrillation

Deviation from NSR
No organized atrial depolarization, so no normal P

waves (impulses are not originating from the sinus node).
Atrial activity is chaotic (resulting in an irregularly irregular rate).
Common, affects 2-4%, up to 5-10% if > 80 years old
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Atrial Fibrillation Etiology: due to multiple re-entrant wavelets conducted between the

Atrial Fibrillation

Etiology: due to multiple re-entrant wavelets conducted between the R

& L atria and the impulses are formed in a totally unpredictable fashion.
The AV node allows some of the impulses to pass through at variable intervals (so rhythm is irregularly irregular).
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Rhythm #6 70 bpm Rate? Regularity? regular flutter waves 0.06 s

Rhythm #6

70 bpm

Rate?

Regularity?

regular

flutter waves

0.06 s

P waves?

PR interval?

none

QRS duration?

Interpretation?

Atrial Flutter

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Atrial Flutter Deviation from NSR No P waves. Instead flutter waves

Atrial Flutter
Deviation from NSR
No P waves. Instead flutter waves (note “sawtooth”

pattern) are formed at a rate of 250 - 350 bpm.
Only some impulses conduct through the AV node (usually every other impulse).
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Atrial Flutter Etiology: Reentrant pathway in the right atrium with every

Atrial Flutter
Etiology: Reentrant pathway in the right atrium with every 2nd,

3rd or 4th impulse generating a QRS (others are blocked in the AV node as the node repolarizes).
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Rhythm #7 74 ?148 bpm Rate? Regularity? Regular ? regular Normal

Rhythm #7

74 ?148 bpm

Rate?

Regularity?

Regular ? regular

Normal ? none

0.08 s

P waves?

PR interval?

0.16 s ? none

QRS duration?

Interpretation?

Paroxysmal Supraventricular Tachycardia
(PSVT)

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PSVT: Paroxysmal Supra Ventricular Tachycardia Deviation from NSR The heart rate

PSVT: Paroxysmal Supra Ventricular Tachycardia
Deviation from NSR
The heart rate suddenly speeds up,

often triggered by a PAC (not seen here) and the P waves are lost.
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AV Nodal Blocks 1st Degree AV Block 2nd Degree AV Block,

AV Nodal Blocks

1st Degree AV Block
2nd Degree AV Block, Type I
2nd

Degree AV Block, Type II
3rd Degree AV Block
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Rhythm #10 60 bpm Rate? Regularity? regular normal 0.08 s P

Rhythm #10

60 bpm

Rate?

Regularity?

regular

normal

0.08 s

P waves?

PR interval?

0.36 s

QRS duration?

Interpretation?

1st Degree AV Block

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1st Degree AV Block Deviation from NSR PR Interval > 0.20 s

1st Degree AV Block
Deviation from NSR
PR Interval > 0.20 s

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1st Degree AV Block Etiology: Prolonged conduction delay in the AV node or Bundle of His.

1st Degree AV Block
Etiology: Prolonged conduction delay in the AV node

or Bundle of His.
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Rhythm #11 50 bpm Rate? Regularity? regularly irregular nl, but 4th

Rhythm #11

50 bpm

Rate?

Regularity?

regularly irregular

nl, but 4th no QRS

0.08 s

P waves?

PR interval?

lengthens

QRS duration?

Interpretation?

2nd Degree AV Block, Type I

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2nd Degree AV Block, Type I Deviation from NSR PR interval

2nd Degree AV Block, Type I
Deviation from NSR
PR interval progressively lengthens,

then the impulse is completely blocked (P wave not followed by QRS).
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2nd Degree AV Block, Type I Etiology: Each successive atrial impulse

2nd Degree AV Block, Type I
Etiology: Each successive atrial impulse encounters

a longer and longer delay in the AV node until one impulse (usually the 3rd or 4th) fails to make it through the AV node.
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Rhythm #12 40 bpm Rate? Regularity? regular nl, 2 of 3

Rhythm #12

40 bpm

Rate?

Regularity?

regular

nl, 2 of 3 no QRS

0.08 s

P waves?

PR interval?

0.14 s

QRS duration?

Interpretation?

2nd Degree AV Block, Type II

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2nd Degree AV Block, Type II Deviation from NSR Occasional P

2nd Degree AV Block, Type II
Deviation from NSR
Occasional P waves are

completely blocked (P wave not followed by QRS).
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Rhythm #13 40 bpm Rate? Regularity? regular no relation to QRS

Rhythm #13

40 bpm

Rate?

Regularity?

regular

no relation to QRS

wide (> 0.12 s)

P waves?

PR interval?

none

QRS duration?

Interpretation?

3rd Degree AV Block

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3rd Degree AV Block Deviation from NSR The P waves are

3rd Degree AV Block
Deviation from NSR
The P waves are completely blocked

in the AV junction; QRS complexes originate independently from below the junction.
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3rd Degree AV Block Etiology: There is complete block of conduction

3rd Degree AV Block
Etiology: There is complete block of conduction in

the AV junction, so the atria and ventricles form impulses independently of each other.
Without impulses from the atria, the ventricles own intrinsic pacemaker kicks in at around 30 - 45 beats/minute.
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Remember When an impulse originates in a ventricle, conduction through the

Remember

When an impulse originates in a ventricle, conduction through the ventricles

will be inefficient and the QRS will be wide and bizarre.
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Ventricular Fibrillation Rhythm: irregular-coarse or fine, wave form varies in size

Ventricular Fibrillation

Rhythm: irregular-coarse or fine, wave form varies in size and

shape
Fires continuously from multiple foci
No organized electrical activity
No cardiac output
Causes: MI, ischemia, untreated VT, underlying CAD, acid base imbalance, electrolyte imbalance, hypothermia,
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Ventricular Tachycardia Ventricular cells fire continuously due to a looping re-entrant

Ventricular Tachycardia

Ventricular cells fire continuously due to a looping re-entrant circuit


Rate usually regular, 100 - 250 bpm
P wave: may be absent, inverted or retrograde
QRS: complexes bizarre, > .12
Rhythm: usually regular
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Asystole Ventricular standstill, no electrical activity, no cardiac output – no

Asystole

Ventricular standstill, no electrical activity, no cardiac output – no pulse!
Cardiac

arrest, may follow VF or PEA
Remember! No defibrillation with Asystole
Rate: absent due to absence of ventricular activity. Occasional P wave may be identified.
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IdioVentricular Rhythm Escape rhythm (safety mechanism) to prevent ventricular standstill HIS/purkinje

IdioVentricular Rhythm

Escape rhythm (safety mechanism) to prevent ventricular standstill
HIS/purkinje system takes

over as the heart’s pacemaker
Treatment: pacing
Rhythm: regular
Rate: 20-40 bpm
P wave: absent
QRS: > .12 seconds (wide and bizarre)
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Diagnosing a MI To diagnose a myocardial infarction you need to

Diagnosing a MI

To diagnose a myocardial infarction you need to go

beyond looking at a rhythm strip and obtain a 12-Lead ECG.

Rhythm Strip

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The 12-Lead ECG The 12-Lead ECG sees the heart from 12

The 12-Lead ECG

The 12-Lead ECG sees the heart from 12 different

views.
Therefore, the 12-Lead ECG helps you see what is happening in different portions of the heart.
The rhythm strip is only 1 of these 12 views.
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The 12-Leads The 12-leads include: 3 Limb leads (I, II, III)

The 12-Leads

The 12-leads include:

3 Limb leads (I, II, III)

3 Augmented leads

(aVR, aVL, aVF)

6 Precordial leads (V1- V6)

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Views of the Heart Some leads get a good view of

Views of the Heart

Some leads get a good view of the:

Anterior

portion of the heart

Lateral portion of the heart

Inferior portion of the heart

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ST Elevation One way to diagnose an acute MI is to

ST Elevation

One way to diagnose an acute MI is to look

for elevation of the ST segment.
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ST Elevation (cont) Elevation of the ST segment (greater than 1

ST Elevation (cont)

Elevation of the ST segment (greater than 1 small

box) in 2 leads is consistent with a myocardial infarction.
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Anterior View of the Heart The anterior portion of the heart

Anterior View of the Heart

The anterior portion of the heart is

best viewed using leads V1- V4.
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Anterior Myocardial Infarction If you see changes in leads V1 -

Anterior Myocardial Infarction

If you see changes in leads V1 - V4

that are consistent with a myocardial infarction, you can conclude that it is an anterior wall myocardial infarction.
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Putting it all Together Do you think this person is having

Putting it all Together

Do you think this person is having a

myocardial infarction. If so, where?
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Interpretation Yes, this person is having an acute anterior wall myocardial infarction.

Interpretation

Yes, this person is having an acute anterior wall myocardial infarction.

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Other MI Locations Now that you know where to look for

Other MI Locations

Now that you know where to look for an

anterior wall myocardial infarction let’s look at how you would determine if the MI involves the lateral wall or the inferior wall of the heart.
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Views of the Heart Some leads get a good view of

Views of the Heart

Some leads get a good view of the:

Anterior

portion of the heart

Lateral portion of the heart

Inferior portion of the heart

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Other MI Locations Second, remember that the 12-leads of the ECG

Other MI Locations

Second, remember that the 12-leads of the ECG look

at different portions of the heart. The limb and augmented leads “see” electrical activity moving inferiorly (II, III and aVF), to the left (I, aVL) and to the right (aVR). Whereas, the precordial leads “see” electrical activity in the posterior to anterior direction.

Limb Leads

Augmented Leads

Precordial Leads

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Other MI Locations Now, using these 3 diagrams let’s figure where

Other MI Locations

Now, using these 3 diagrams let’s figure where to

look for a lateral wall and inferior wall MI.

Limb Leads

Augmented Leads

Precordial Leads

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Anterior MI Remember the anterior portion of the heart is best

Anterior MI

Remember the anterior portion of the heart is best viewed

using leads V1- V4.

Limb Leads

Augmented Leads

Precordial Leads

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Lateral MI So what leads do you think the lateral portion

Lateral MI

So what leads do you think the lateral portion of

the heart is best viewed?

Limb Leads

Augmented Leads

Precordial Leads

Leads I, aVL, and V5- V6

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Inferior MI Now how about the inferior portion of the heart?

Inferior MI

Now how about the inferior portion of the heart?

Limb

Leads

Augmented Leads

Precordial Leads

Leads II, III and aVF

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Putting it all Together Now, where do you think this person is having a myocardial infarction?

Putting it all Together

Now, where do you think this person is

having a myocardial infarction?
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Inferior Wall MI This is an inferior MI. Note the ST

Inferior Wall MI

This is an inferior MI. Note the ST elevation

in leads II, III and aVF.
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Putting it all Together How about now?

Putting it all Together

How about now?

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Anterolateral MI This person’s MI involves both the anterior wall (V2-V4)

Anterolateral MI

This person’s MI involves both the anterior wall (V2-V4) and

the lateral wall (V5-V6, I, and aVL)!
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Reading 12-Lead ECGs The best way to read 12-lead ECGs is

Reading 12-Lead ECGs

The best way to read 12-lead ECGs is to

develop a step-by-step approach (just as we did for analyzing a rhythm strip). In these modules we present a 6-step approach:
Calculate RATE
Determine RHYTHM
Determine QRS AXIS
Calculate INTERVALS
Assess for HYPERTROPHY
Look for evidence of INFARCTION
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Rate Rhythm Axis Intervals Hypertrophy Infarct In Module II you learned

Rate Rhythm Axis Intervals Hypertrophy Infarct

In Module II you learned

how to calculate the rate. If you need a refresher return to that module.
There is one new thing to keep in mind when determining the rate in a 12-lead ECG…